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Dysregulation of pulmonary endothelial protein C receptor and thrombomodulin in severe falciparum malaria-associated ARDS relevant to hemozoin - Fig 2
Abstract
<p><b>Immunohistochemical staining of EPCR in the normal (A), non-PE (B), PE (C) and ARDS (D) lungs</b>. The EPCR expression in small blood vessels are shown in the inset of each micrograph. The H-score expression is demonstrated by bar graphs for PE (E) or ARDS (F) lungs, to compare those expression Friedman test was performed (**; p-value < 0.001, ***; p-value <0.0001). Negative correlation between the level of EPCR and some histopathological changes was observed using Spearman test (G-I).</p- Image
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- Hematology
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- apoptotic pathway
- electron microscopy
- protein C receptor
- caspase recruitment domain -9 messenger-ribonucleic acid
- ARDS patients
- proinflammatory cytokines-induced type II pneumocyte injury
- 29 left-over lung specimens
- pneumocytic cell injury
- blood cell accumulation
- interleukin -13
- blood cell
- EB
- hemozoin deposition
- falciparum malaria
- PRBC
- protein C system
- distress syndrome
- ARDS coexisted
- EPCR
- TM
- falciparum malaria-associated ARDS
- malaria-associated ARDS
- CARD -9 mRNA